TREATING STROKE AND MOTOR NEURONE DISEASE USING OMI THERAPY
Even in developed countries, brain catastrophe is the third-leading cause of death and is responsible for a significant majority of disabilities. In 90 % of cases, damage to the cerebral matter is triggered by vas ospasm, with hemorrhages being responsible for the other 10 %. When this happens, the entire brain region exhibiting signs of neurological deficit does not die immediately and the functioning of the penumbra or “sleeping” parts of the brain may still be saved if the right methods are employed. The effectiveness of conventional medical therapy can be significantly improved using OMI treatment. Given optimal nerve cell metabolism and a reduction in the temporary diffuse or localized edema brought about by the illness, OMI therapy can, if used during the acute phase, help reduce the secondary damage caused by intracranial pressure.
As a neurologist and someone who performs carotid ultrasound examinations, I come across many acute and chronically ill patients. Four years ago, I became familiar with OMI therapy and, since then, have been using it confidently and successfully to treat my patients. I would like to present some case studies to those interested.
D.: This 15-year old boy was intubated by the emergency services after a night out and supplied with a peripheral venous catheter in his right arm by the traumatology team after collapsing down at home and entering into a state of shock. It could neither be confirmed nor ruled out that the boy had taken drugs. He had no external injuries and his pupils were small. The patient‘s left limbs were limp and lame while the limbs on the right side moved only sluggishly in response to pain stimulation. The urgently performed cranial CT showed neither brain edema nor any other abnormality and so the patient was admitted by the neurology/toxicology teams. By evening time, brain stem symptoms (intermittent vertical nystagmus) was determined. The control cranial CT carried out the next day showed extended malacia in the brain stem (bridges) and malacia 10 mm in size in the patient‘s cerebellum. The carotid did not display any UH abnormality. In addition to the abnormality mentioned above, the cranial MR and MR angiography both confirmed the occlusion of a segment of 8 mm in front of the basilar origin. On the patient‘s third day in hospital, his mother came to seen us, requesting OMI therapy for her son, which we began the following day using the OMI THERAPY Plus.The treatment was administered three times a day (levels 3, 10 and P1 on the mat and P4 using the cushion placed under the patient‘s head). To begin with, the patient became more alert, opening his eyes both spontaneously and when prompted. Then, he began to voluntarily move: first his right hand, followed by his left leg. The breathing and feeding tubes were removed towards the end of the fourth week. The patient‘s ability to swallow gradually improved while communication improved too, despite the presence of motor aphasia.
On the sixth day of OMI treatment, the control cranial MR showed an improvement in basilar filling.
Gy. S. C.: This 46-year old man was admitted to the neurology department after suffering for one week with headaches, vomiting and impaired vision, which were not alleviated by pain medication. Upon admission, the patient did not display any focal signs. The urgent cranial CT did not show up any abnormalities but the blood content of the cerebrospinal fluid taken with the lumbar puncture did show subarachnoid bleeding while the cranial MR and MR angiography indicated two small aneurysms of the anterior com¬municating artery. During neurosurgery, a remedy was supplied using the endovascular method. Afterwards, in addition to occipital binding, the patient became increasingly disoriented with only sluggish movement of the limbs in response to pain stimulation. The cranial CT did not detect any bleeding but the anterior cerebral artery on both sides did indicate localized circulatory disturbance. An MRSA infection made his condition deteriorate further, and bedsores developed too. Although his condition improved as a result of rehabilitative treatment, upon leaving for home, he could not move his legs and was doubly incontinent. Seven months after the onset of his illness, we began treating the patient with OMI Classic (programs 3 and P1 on the mat in the first week, from week two programs 3 and P1 on the mat, from the third week programs 3 and P1 on the mat and, because of shoulder pain, program P3 with the Ring). In the second week of treatment, he was doubly continent during the day. In the third week, he was able to bend his legs well while, in week four, he was able to lift his legs right from the hip. Amyotrophic lateral sclerosis is a rare progressive illness where the central movement cells die off. Based on current scientific knowledge, the disease is incurable. The symptoms are the wastage and weakening of muscles, first in typical areas and then throughout the entire body, unwanted muscle spasms and, later, problems swallowing and breathing. L. Z.: A 55-year old patient who has been treated for schizophrenia and parkinsonism since his youth. Ap¬proximately one year ago, his mother noticed that he was holding pens and spoons in an unusual manner. Because of his underlying illness, she did not attach any significance to this. However, for some months, he was increasingly asking for help in buttoning his shirt and tying his shoelaces.
Observation status: Limp muscles throughout the patient‘s body. Muscle wastage between the first and second fingers on each hand and in the shoulder girdles. Subtle, yet frequent, muscle spasms in the muscles of the shoulder girdle. Ability to press and form a circle with the hand is becoming more and more diminished.
His laboratory findings did not indicate any abnormalities. No MR abnormalities in the skull or cervical spine. ENG investigation confirmed motor neurone disease. We began ambulatory treatment with the OMI Professional device three to four times per week (mat P1 and 1). After two weeks of treatment, the patient‘s mother reported that his night-time urination problem had ceased. After two months of treatment, the patient‘s ability to grasp things with his right hand improved, his appetite improved and his anxiety subsided – even though visual examination did not show any change in terms of muscle mass or wastage.